KLEIBERIT PRIMER 838.0
Flammability | 3 | |
Toxicity | 2 | |
Body Contact | 2 | |
Reactivity | 1 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
The use of a quantity of material in an unventilated or confined space may result in
increased exposure and an irritating atmosphere developing.Before starting consider
control of exposure by mechanical ventilation. A primer used to improve the adhesion of
EVA based hot melt adhesives to PVC and polyester edges, acrylic resin edges, solid wood
edges and HPL edging strips. Application is by means of a brush.
"primer for eva hot melt adhesives"
HARMFUL - May cause lung damage if swallowed.
Irritating to eyes and respiratory system.
Highly flammable.
Repeated exposure may cause skin dryness and cracking.
Vapors may cause dizziness or suffocation.
Accidental ingestion of the material may be damaging to the health of the individual. Swallowing of the liquid may cause aspiration into the lungs with the risk of chemical pneumonitis; serious consequences may result. (ICSC13733).
There is evidence that material may produce eye irritation in some persons and produce eye damage 24 hours or more after instillation. Severe inflammation may be expected with pain. There may be damage to the cornea. Unless treatment is prompt and adequate there may be permanent loss of vision. Conjunctivitis can occur following repeated exposure. The vapour when concentrated has pronounced eye irritation effects and this gives some warning of high vapour concentrations. If eye irritation occurs seek to reduce exposure with available control measures, or evacuate area. The liquid may produce eye discomfort and is capable of causing temporary impairment of vision and/or transient eye inflammation, ulceration.
Skin contact with the material may damage the health of the individual; systemic effects may result following absorption. There is some evidence to suggest that the material may cause moderate inflammation of the skin either following direct contact or after a delay of some time. Repeated exposure can cause contact dermatitis which is characterized by redness, swelling and blistering. Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected. The material may cause skin irritation after prolonged or repeated exposure and may produce on contact skin redness, swelling, the production of vesicles, scaling and thickening of the skin.
Inhalation may produce health damage*. Inhalation of vapors or aerosols (mists, fumes), generated by the material during the course of normal handling, may be damaging to the health of the individual. The material can cause respiratory irritation in some persons. The body's response to such irritation can cause further lung damage. Systemic effects of acetone inhalation exposure include central nervous system depression, light-headedness, incoherent speech, ataxia, stupor, hypotension, tachycardia, metabolic acidosis, hyperglycaemia and ketosis. Rarely, convulsions and tubular necrosis may be evident. Other symptoms of exposure may include restlessness, headache, vomiting, low blood-pressure and rapid and irregular pulse, eye and throat irritation, weakness of the legs and dizziness. Inhalation of high concentrations may produce dryness of the mouth and throat, nausea, uncoordinated movement, loss of coordinated speech, drowsiness and, in severe cases, coma. Inhalation of acetone vapours over long periods causes irritation of the respiratory tract, coughing and headache. Rats exposed to 52200 ppm vapour for 1 hour showed clear signs of narcosis; fatalities occurred at 126600 ppm. If exposure to highly concentrated solvent atmosphere is prolonged this may lead to narcosis, unconsciousness, even coma and possible death.
Limited evidence suggests that repeated or long-term occupational exposure may produce cumulative health effects involving organs or biochemical systems. Asthma-like symptoms may continue for months or even years after exposure to the material ceases. This may be due to a non-allergenic condition known as reactive airways dysfunction syndrome (RADS) which can occur following exposure to high levels of highly irritating compound. Key criteria for the diagnosis of RADS include the absence of preceding respiratory disease, in a non-atopic individual, with abrupt onset of persistent asthma-like symptoms within minutes to hours of a documented exposure to the irritant. A reversible airflow pattern, on spirometry, with the presence of moderate to severe bronchial hyperreactivity on methacholine challenge testing and the lack of minimal lymphocytic inflammation, without eosinophilia, have also been included in the criteria for diagnosis of RADS. RADS (or asthma) following an irritating inhalation is an infrequent disorder with rates related to the concentration of and duration of exposure to the irritating substance. Industrial bronchitis, on the other hand, is a disorder that occurs as result of exposure due to high concentrations of irritating substance (often particulate in nature) and is completely reversible after exposure ceases. The disorder is characterised by dyspnea, cough and mucus production. Workers exposed to 700 ppm acetone for 3 hours/day for 7-15 years showed inflammation of the respiratory tract, stomach and duodenum, attacks of giddiness and loss of strength. Exposure to acetone may enhance liver toxicity of chlorinated solvents.