UNIMIN PYROPHYLLITE
Flammability | 0 | |
Toxicity | 2 | |
Body Contact | 2 | |
Reactivity | 0 | |
Chronic | 0 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Used e.g. in refractories, ceramics, plastics, paints and adhesives.
"hydrous aluminium silicate"
Harmful: danger of serious damage to health by prolonged exposure through
inhalation.
Although ingestion is not thought to produce harmful effects, the material may still be damaging to the health of the individual following ingestion, especially where pre- existing organ (e.g. liver, kidney) damage is evident. Present definitions of harmful or toxic substances are generally based on doses producing mortality (death) rather than those producing morbidity (disease, ill-health). Gastrointestinal tract discomfort may produce nausea and vomiting. In an occupational setting however, ingestion of insignificant quantities is not thought to be cause for concern. Considered an unlikely route of entry in commercial/industrial environments. Considered to be non toxic.
Although the material is not thought to be an irritant, direct contact with the eye may produce transient discomfort characterized by tearing or conjunctival redness (as with windburn). The dust may produce eye discomfort and abrasive eye inflammation.
The material is not thought to produce adverse health effects or skin irritation following contact (as classified using animal models). Nevertheless, good hygiene practice requires that exposure be kept to a minimum and that suitable gloves be used in an occupational setting.
The material is not thought to produce adverse health effects or irritation of the respiratory tract (as classified using animal models). Nevertheless, good hygiene practice requires that exposure be kept to a minimum and that suitable control measures be used in an occupational setting. Persons with impaired respiratory function, airway diseases and conditions such as emphysema or chronic bronchitis, may incur further disability if excessive concentrations of particulate are inhaled. Effects on lungs are significantly enhanced in the presence of respirableparticles. Acute silicosis occurs under conditions of extremely high silica dust exposure particularly when the particle size of the dust is small. The disease is rapidly progressive and spreads widely through the lungs within months of the initial exposure and causing deaths within 1 to 2 years.
Principal routes of exposure are by accidental skin and eye contact andinhalation of generated dusts. Crystalline silicas activate the inflammatory response of white blood cells after they injure the lung epithelium. Chronic exposure to crystalline silicas reduce lung capacity and predispose to chest infections. A large part of the crystals accumulates in the lungs. Silicosis can occur, a condition where irreversible scarring of the lung occurs. Symptoms do not appear until months to years after exposure. Smoking increases this risk. Most simple cases of silicosis do not produce symptoms, but they can progress and eventually cause a tuberculosis-like syndrome which can be fatal. When silicosis is advanced, there is an increased risk of lung cancer and lymphoma. Laws in some areas require those exposed to silica to be under health surveillance.