Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

K&H 350G CONTACT ADHESIVE (AEROSOL)

NFPA

PRODUCT USE

The use of a quantity of material in an unventilated or confined space may result in
increased exposure and an irritating atmosphere developing.Before starting consider
control of exposure by mechanical ventilation. Application is by spray atomization from a
hand held aerosol pack. Adhesives.

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

Harmful if swallowed.
Irritating to skin.
Limited evidence of a carcinogenic effect.
Possible risk of impaired fertility.
Harmful: danger of serious damage to health by prolonged exposure through
inhalation.
Extremely flammable.
Vapors may cause dizziness or suffocation.
Toxic to aquatic organisms, may cause long- term adverse effects in the aquatic
environment.
Risk of explosion if heated under confinement.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual.  Swallowing of the liquid may cause aspiration into the lungs with the risk of chemical pneumonitis; serious consequences may result. (ICSC13733).  

EYE

  Not considered to be a risk because of the extreme volatility of the gas.  There is some evidence that material may produce eye irritation in some persons and produce eye damage 24 hours or more after instillation. Moderate inflammation may be expected with redness; conjunctivitis may occur with prolonged exposure.  

SKIN

  The material may accentuate any pre-existing dermatitis condition.  Skin contact with the material may damage the health of the individual; systemic effects may result following absorption.  Spray mist may produce discomfort.  The liquid may be miscible with fats or oils and may degrease the skin, producing a skin reaction described as non-allergic contact dermatitis. The material is unlikely to produce an irritant dermatitis as described in EC Directives .  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  The material may cause moderate inflammation of the skin either following direct contact or after a delay of some time. Repeated exposure can cause contact dermatitis which is characterized by redness, swelling and blistering.  

INHALED

  Inhalation may produce health damage*.  Inhalation of vapours may cause drowsiness and dizziness. This may be accompanied by narcosis, reduced alertness, loss of reflexes, lack of coordination and vertigo.  Inhalation of aerosols (mists, fumes), generated by the material during the course of normal handling, may be damaging to the health of the individual.  Inhaling high concentrations of mixed hydrocarbons can cause narcosis, with nausea, vomiting and lightheadedness. Low molecular weight (C2-C12) hydrocarbons can irritate mucous membranes and cause incoordination, giddiness, nausea, vertigo, confusion, headache, appetite loss, drowsiness, tremors and stupor. Massive exposures can lead to severe central nervous system depression, deep coma and death. Convulsions can occur due to brain irritation and/or lack of oxygen. Permanent scarring may occur, with epileptic seizures and brain bleeds occurring months after exposure. Respiratory system effects include inflammation of the lungs with edema and bleeding. Lighter species mainly cause kidney and nerve damage; the heavier paraffins and olefins are especially irritant to the respiratory system. Alkenes produce pulmonary edema at high concentrations. Liquid paraffins may produce sensation loss and depressant actions leading to weakness, dizziness, slow and shallow respiration, unconsciousness, convulsions and death. C5-7 paraffins may also produce multiple nerve damage. Aromatic hydrocarbons accumulate in lipid rich tissues (typically the brain, spinal cord and peripheral nerves) and may produce functional impairment manifested by nonspecific symptoms such as nausea, weakness,  fatigue, vertigo; severe exposures may produce inebriation or unconsciousness. Many of the petroleum hydrocarbons can sensitize the heart and may cause ventricular fibrillation,  leading to death.  Central nervous system (CNS) depression may include general discomfort, symptoms of giddiness, headache, dizziness, nausea, anaesthetic effects, slowed reaction time, slurred speech and may progress to unconsciousness. Serious poisonings may result in respiratory depression and may be fatal.  Material is highly volatile and may quickly form a concentrated atmosphere in confined or unventilated areas. Vapor is heavier than air and may displace and replace air in breathing zone, acting as a simple asphyxiant. This may happen with little warning of overexposure.  Inhalation of high concentrations of gas/vapor causes lung irritation with coughing and nausea, central nervous depression with headache and dizziness, slowing of reflexes, fatigue and inco-ordination.  If exposure to highly concentrated solvent atmosphere is prolonged this may lead to narcosis, unconsciousness, even coma and possible death.  WARNING: Intentional misuse by concentrating/inhaling contents may be lethal.  Inhalation exposure may cause susceptible individuals to show change in heart beat rhythm i.e. cardiac arrhythmia. Exposures must be terminated.  Acute intoxication by halogenated aliphatic hydrocarbons appears to take place over two stages. Signs of a reversible narcosis are evident in the first stage and in the second stage signs of injury to organs may become evident, a single organ alone is (almost) never involved.  At high concentrations most of the absorbed methylene chloride (dichloromethane) is exhaled unchanged; the remainder is metabolised to carbon monoxide, carbon dioxide and inorganic chloride. Inhalation may produce fatigue, weakness, sleepiness, light-  headedness, chills, nausea, diarrhoea and abdominal pain. The lowest published lethal dose is 20,000 ppm for 20 hours. The body metabolises methylene chloride to carbon monoxide and adds to the body burden of carboxyhaemoglobin (COHb) contributed by other sources. The increase in COHb is related to the magnitude of vapour exposure and duration. Serious poisoning can occur without raised COHb concentrations, although these raised concentrations may persist for several hours. Central nervous system (CNS) effects are thought to be due to methylene chloride itself or methylene chloride in combination with other sources of COHb, rather than the COHb metabolite. The raised COHb concentrations are not usually expected to produce adverse effects in healthy individuals but may be cause for concern in individuals with cardiovascular disease. Encephalopathy (brain injury) has been reported after repeated exposure. Angina, myocardial infarction, cardiac arrhythmias and cardiac arrest have also been reported, although the cardiovascular system is not generally a target for methylene chloride toxicity. Hypotension, shock and metabolic acidosis may also occur as a result of overexposure. Respiratory failure may develop, secondary to CNS depression, in severe cases.  

CHRONIC HEALTH EFFECTS

  Harmful: danger of serious damage to health by prolonged exposure through inhalation.  Harmful: danger of serious damage to health by prolonged exposure through inhalation.  This material can cause serious damage if one is exposed to it for long periods. It can be assumed that it contains a substance which can produce severe defects. This has been demonstrated via both short- and long-term experimentation.  Ample evidence from experiments exists that there is a suspicionthis material directly reduces fertility.  Principal route of occupational exposure to the gas is by inhalation.  Constant or exposure over long periods to mixed hydrocarbons may produce stupor with dizziness, weakness and visual disturbance, weight loss and anemia, and reduced liver and kidney function. Skin exposure may result in drying and cracking and redness of the skin. Chronic exposure to lighter hydrocarbons can cause nerve damage, peripheral neuropathy, bone marrow dysfunction and psychiatric disorders as well as damage the liver and kidneys.  Chronic inhalation or skin exposure to n-hexane may cause damage to nerve ends in extremities, e.g. finger, toes with loss of sensation. Symptoms can progress for months even after removal of exposure, and recovery may take years and may not be complete.  Dichloromethane exposures cause liver and kidney damage in animals and this justifies consideration before exposing persons with a history of impaired liver function and/or renal disorders.