Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

HR SIMON CHEMBLEND2 DEVELOPER PART A

NFPA

PRODUCT USE

Used according to manufacturer' s directions. Requires that the two parts be mixed by hand
or mixer before use, in accordance with manufacturers directions. Mix only as much as is
required. Do not return the mixed material to the original containers.

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

Harmful if swallowed.
Contact with acids liberates toxic gas.
May cause SENSITIZATION by skin contact.
Limited evidence of a carcinogenic effect.
Possible risk of impaired fertility.
Possible risk of harm to the unborn child.
Possible risk of irreversible effects.
Irritating to eyes, respiratory system and skin.
Toxic to aquatic organisms.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual.  Ingestion of sulfite salts may cause gastric irritation. Large doses may produce violent colic, diarrhea, circulatory disturbance, depression of vital functions and, sometimes, death.  

EYE

  This material can cause eye irritation and damage in some persons.  

SKIN

  This material can cause inflammation of the skin oncontact in some persons.  The material may accentuate any pre-existing dermatitis condition.  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  

INHALED

  The material can cause respiratory irritation in some persons. The body's response to such irritation can cause further lung damage.  Inhalation of aerosols (mists, fumes), generated by the material during the course of normal handling, may be damaging to the health of the individual.  Sulfur dioxide irritation probably results from the action of sulfurous acid as the highly soluble gas dissolves in mucous fluid. Short-term exposure causes bronchoconstriction measurable as an increase in flow-resistance. The magnitude is concentration-dependent..  Chief effects are upper respiratory tract irritation and severe acute exposure may cause oedema of the lungs and possible respiratory paralysis. These exposures have produced severe obstructive and restrictive defects up to 3 months post-exposure; these have failed to respond to bronchodilators. Such exposures have also, on rare occasions, been associated with moderately severe obstructive illness and persistent, productive cough.  Systemic effects of acute poisoning are not known but regular exposure may deaden the sense of smell.  Symptoms include throat irritation, coughing, tightness of chest, difficulty with breathing, tear formation (lachrymation), eye smarting and suffocating feeling.  Substantial exposures produce direct respiratory tract irritation, cough, burning, lachrymation, conjunctival injection, difficulty in swallowing, and otopharyngeal erythema. Other symptoms may include vomiting, diarrhoea, abdominal pain, fever, headache,  vertigo, agitation, tremor, convulsions, and peripheral neuritis. High dose acute exposure may produce immediate bronchospasm and pulmonary oedema with respiratory failure/ paralysis, inflammation of the conjunctivae and inflammation of the tongue.  

CHRONIC HEALTH EFFECTS

  There has been concern that this material can cause cancer or mutations, but there is not enough data to make an assessment.  Skin contact with the material is more likely to cause a sensitization reaction in some persons compared to the general population.  Exposure to the material may result in a possible risk of irreversible effects. The material may produce mutagenic effects in man. This concern is raised, generally, on the basis ofappropriate studies using mammalian somatic cells in vivo. Such findings are often supported by positive results from in vitro mutagenicity studies.  Limited evidence suggests that repeated or long-term occupational exposure may produce cumulative health effects involving organs or biochemical systems.  There is some evidence that inhaling this product is more likely to cause a sensitization reaction in some persons compared to the general population.  Repeated or prolonged exposure to corrosives may result in the erosion of teeth, inflammatory and ulcerative changes in the mouth and necrosis (rarely) of the jaw. Bronchial irritation, with cough, and frequent attacks of bronchial pneumonia may ensue. Gastrointestinal disturbances may also occur. Chronic exposures may result in dermatitis and/or conjunctivitis.  Sulfites and bisulfites can cause narrowing of the airways, stomach upset, flushing, low blood pressure. tingling sensation, itchy wheal, swelling and shock, and asthmatics are especially prone. They induce allergic-like reactions which can occur on first contact with the material.  Repeated exposure of animals to airborne sulfur dioxide (SO2) can produce a thickening of the mucous layer in the trachea and an increase in goblet cells and mucous glands similar to pathological changes found in chronic human bronchitis.  Chronic exposure to sulfur dioxide (SO2) particulate complexes, present in polluted air, have been associated with the aggravation of chronic cardiovascular diseases such as asthma, chronic pulmonary disease, and coronary artery disease (this may occur at levels of 6-10 ug/m3 for 24 hours), An association exists between persistent cough and sputum production, particularly in women and non-smokers. A 10-year follow study on workers exposed to a mean sulfur dioxide concentration of up to 33 ppm did not reveal an increased prevalence of chronic respiratory disease or decreased pulmonary function.  By contrast, studies of smelter workers, exposed to concentrations below 2 ppm, suggest that chronic respiratory disease may develop and that workers exposed at concentrations exceeding 1 ppm show accelerated loss of pulmonary function.  Although SO2 is not a carcinogen, the apparent increases in mortalities amongst arsenic-  exposed smelter workers was greater when exposures included both high arsenic concentrations and moderate to high SO2 exposures, suggesting that SO2 might act as a promoter.  Intermittent exposure of rats to benz[a]pyrene along with inhalation of SO2 at 4-10 ppm, 1-6 hours per day, 5 days per week, produced substantial increases in respiratory tract squamous cell carcinomas compared to that associated with exposure to B[a]P or SO2 alone.