Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

K&H GEL COAT REPAIR PUTTY

NFPA

PRODUCT USE

Used for the general repairs of metal, masonry, wooden and fibreglass surfaces. The use of
a quantity of material in an unventilated or confined space may result in increased
exposure and an irritating atmosphere developing.Before starting consider control of
exposure by mechanical ventilation.

SYNONYMS

"repair putty"

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

Limited evidence of a carcinogenic effect.
Harmful by inhalation and if swallowed.
Irritating to eyes and skin.
Flammable.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual.  Considered an unlikely route of entry in commercial/industrial environments.  Ingestion may result in nausea, abdominal irritation, pain and vomiting.  

EYE

  This material can cause eye irritation and damage in some persons.  The vapour when concentrated has pronounced eye irritation effects and this gives some warning of high vapour concentrations. If eye irritation occurs seek to reduce exposure with available control measures, or evacuate area.  The material may produce severe irritation to the eye causing pronounced inflammation. Repeated or prolonged exposure to irritants may produce conjunctivitis.  

SKIN

  This material can cause inflammation of the skin oncontact in some persons.  Skin contact is not thought to have harmful health effects, however the material may still produce health damage following entry through wounds, lesions or abrasions.  Bare unprotected skin should not be exposed to this material.  The material may accentuate any pre-existing dermatitis condition.  The material may cause skin irritation after prolonged or repeated exposure and may produce on contact skin redness, swelling, the production of vesicles, scaling and thickening of the skin.  

INHALED

  The material is not thought to produce respiratory irritation (as classified using animal models). Nevertheless inhalation of the material, especially for prolonged periods, may produce respiratory discomfort and occasionally, distress.  Inhalation hazard is increased at higher temperatures.  Acute effects from inhalation of high vapor concentrations may be chest and nasal irritation with coughing, sneezing, headache and even nausea.  If exposure to highly concentrated vapor atmosphere is prolonged this may lead to narcosis, unconsciousness, even coma and unless resuscitated - death.  Inhalation of vapor may aggravate a pre-existing respiratory condition.  Central nervous system (CNS) depression is seen at styrene exposures exceeding 50 ppm, whilst headache, fatigue, nausea and dizziness are reported consistently at exposures of 100 ppm.  Eye and throat irritation occurred in human volunteers exposed to 376 ppm styrene for 1 hour and was accompanied by increased nasal secretion at exposures of 800 ppm for 4 hours. At the end of an 8-hour workshift, workers exposed to 212 ppm styrene had higher urinary levels of alanine-aminopeptidase and N-acetyl-glucosaminidase than unexposed workers, indicating potential renal effects of styrene .  Evidence exists that 5% to 10% reductions in sensory nerve conduction occur at 100 ppm and that slowed reaction times occur after exposure to 50 ppm. Exposure at 370 ppm produces unpleasant subjective symptoms and signs of neurological impairment. High vapour concentrations may have a toxic and anaesthetic effect which may lead to unconsciousness or death. Exposure at 1000 ppm can rapidly lead to unconsciousness whilst exposure to 10000 ppm may cause death in less than one hour. Simple reaction times were increased and coordination decreased amongst volunteers inhaling 350 ppm (via mouth tube) for 30 minutes. Controlled inhalation studies with 300 ppm (via mouth tube) for one hour found reduced ocular tracking abilities but no changes in balance or coordination.  In humans exposed to styrene vapor, pulmonary retention is approximately 66% of the administered concentration.  Following inhalation exposure, styrene is preferentially distributed to adipose tissue. Fat levels in rats were 10-times greater than levels in observed organs after exposure to 50-2000 ppm for 5 hours.  Urinary excretion is the major route of elimination of styrene. In humans, the main urinary metabolites are mandelic acid and phenylglyoxylic acid; rats also excrete hippuric acid and glucuronide. Human volunteers exposed by inhalation to 50 to 200 parts per million (ppm) showed biphasic urinary elimination of mandelic acid with a half-life for the first phase of 4 hours and for the second phase of 25 hours. Urinary metabolite concentrations have been correlated with exposure concentrations in humans.  

CHRONIC HEALTH EFFECTS

  There has been concern that this material can cause cancer or mutations, but there is not enough data to make an assessment.  

  Principal routes of exposure are usually by skin contact with liquid and inhalation of vapor.  Prolonged or continuous skin contact with the liquid may cause defatting with drying, cracking, irritation and dermatitis following.