Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

WAPROO STRIPPER

NFPA

PRODUCT USE

The use of a quantity of material in an unventilated or confined space may result in
increased exposure and an irritating atmosphere developing.Before starting consider
control of exposure by mechanical ventilation. Solvent for cleaning and preparing leather
surfaces before dying / treatment.

SYNONYMS

solvent, cleaning, preparing, leather, dying, treatment

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

May form explosive peroxides.
Harmful if swallowed.
May cause CANCER.
Possible risk of harm to the unborn child.
HARMFUL - May cause lung damage if swallowed.
Harmful: danger of serious damage to health by prolonged exposure through
inhalation.
Irritating to eyes, respiratory system and skin.
Highly flammable.
Vapors may cause dizziness or suffocation.
Toxic to aquatic organisms, may cause long- term adverse effects in the aquatic
environment.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual.  Swallowing of the liquid may cause aspiration into the lungs with the risk of chemical pneumonitis; serious consequences may result. (ICSC13733).  Ingestion of petroleum hydrocarbons can irritate the pharynx, esophagus, stomach and small intestine, and cause swellings and ulcers of the mucous. Symptoms include a burning mouth and throat; larger amounts can cause nausea and vomiting, narcosis, weakness, dizziness, slow and shallow breathing, abdominal swelling, unconsciousness and convulsions. Damage to the heart muscle can produce heart beat irregularities, ventricular fibrillation (fatal) and ECG changes. The central nervous system can be depressed. Light species can cause a sharp tingling of the tongue and cause loss of sensation there. Aspiration can cause cough, gagging, pneumonia with swelling and bleeding.  Following ingestion, a single exposure to isopropyl alcohol produced lethargy and non-  specific effects such as weight loss and irritation. Ingestion of near-lethal doses of isopropanol produces histopathological changes of the stomach, lungs and kidneys, incoordination, lethargy, gastrointestinal tract irritation, and inactivity or anaesthesia.  Swallowing 10 ml. of isopropanol may cause serious injury; 100 ml. may be fatal if not promptly treated. The adult single lethal doses is approximately 250 ml. The toxicity of isopropanol is twice that of ethanol and the symptoms of intoxication appear to be similar except for the absence of an initial euphoric effect; gastritis and vomiting are more prominent. Ingestion may cause nausea, vomiting, and diarrhoea.  There is evidence that a slight tolerance to isopropanol may be acquired.  Considered an unlikely route of entry in commercial/industrial environments. The liquid may produce gastrointestinal discomfort and may be harmful if swallowed. Ingestion may result in nausea, pain and vomiting. Vomit entering the lungs by aspiration may cause potentially lethal chemical pneumonitis.  

EYE

  Direct eye contact with petroleum hydrocarbons can be painful, and the corneal epithelium may be temporarily damaged. Aromatic species can cause irritation and excessive tear secretion.  Isopropanol vapour may cause mild eye irritation at 400 ppm. Splashes may cause severe eye irritation, possible corneal burns and eye damage. Eye contact may cause tearing or blurring of vision.  The liquid produces a high level of eye discomfort and is capable of causing pain and severe conjunctivitis. Corneal injury may develop, with possible permanent impairment of vision, if not promptly and adequately treated.  There is evidence that material may produce eye irritation in some persons and produce eye damage 24 hours or more after instillation. Severe inflammation may be expected with pain. There may be damage to the cornea. Unless treatment is prompt and adequate there may be permanent loss of vision. Conjunctivitis can occur following repeated exposure.  Direct contact of the eye with ethanol may cause immediate stinging and burning with reflex closure of the lid and tearing, transient injury of the corneal epithelium and hyperaemia of the conjunctiva. Foreign-body type discomfort may persist for up to 2 days but healing is usually spontaneous and complete.  

SKIN

  The material may cause moderate inflammation of the skin either following direct contact or after a delay of some time. Repeated exposure can cause contact dermatitis which is characterized by redness, swelling and blistering.  Skin contact with the material may damage the health of the individual; systemic effects may result following absorption.  Open cuts, abraded or irritated skin should not be exposed to this material.  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  Aromatic hydrocarbons may produce sensitivity and redness of the skin. They are not likely to be absorbed into the body through the skin but branched species are more likely to.  

INHALED

  Inhalation may produce health damage*.  The material can cause respiratory irritation in some persons. The body's response to such irritation can cause further lung damage.  Inhalation of vapours may cause drowsiness and dizziness. This may be accompanied by narcosis, reduced alertness, loss of reflexes, lack of coordination and vertigo.  Inhalation hazard is increased at higher temperatures.  Inhaling high concentrations of mixed hydrocarbons can cause narcosis, with nausea, vomiting and lightheadedness. Low molecular weight (C2-C12) hydrocarbons can irritate mucous membranes and cause incoordination, giddiness, nausea, vertigo, confusion, headache, appetite loss, drowsiness, tremors and stupor. Massive exposures can lead to severe central nervous system depression, deep coma and death. Convulsions can occur due to brain irritation and/or lack of oxygen. Permanent scarring may occur, with epileptic seizures and brain bleeds occurring months after exposure. Respiratory system effects include inflammation of the lungs with edema and bleeding. Lighter species mainly cause kidney and nerve damage; the heavier paraffins and olefins are especially irritant to the respiratory system. Alkenes produce pulmonary edema at high concentrations. Liquid paraffins may produce sensation loss and depressant actions leading to weakness, dizziness, slow and shallow respiration, unconsciousness, convulsions and death. C5-7 paraffins may also produce multiple nerve damage. Aromatic hydrocarbons accumulate in lipid rich tissues (typically the brain, spinal cord and peripheral nerves) and may produce functional impairment manifested by nonspecific symptoms such as nausea, weakness,  fatigue, vertigo; severe exposures may produce inebriation or unconsciousness. Many of the petroleum hydrocarbons can sensitize the heart and may cause ventricular fibrillation,  leading to death.  Central nervous system (CNS) depression may include general discomfort, symptoms of giddiness, headache, dizziness, nausea, anaesthetic effects, slowed reaction time, slurred speech and may progress to unconsciousness. Serious poisonings may result in respiratory depression and may be fatal.  Inhalation of high concentrations of gas/vapor causes lung irritation with coughing and nausea, central nervous depression with headache and dizziness, slowing of reflexes, fatigue and inco-ordination.  The odour of isopropanol may give some warning of exposure, but odour fatigue may occur. Inhalation of isopropanol may produce irritation of the nose and throat with sneezing, sore throat and runny nose. The effects in animals subject to a single exposure, by inhalation, included inactivity or anaesthesia and histopathological changes in the nasal canal and auditory canal.  Inhalation of aerosols (mists, fumes), generated by the material during the course of normal handling, may be damaging to the health of the individual.  Ketone vapors irritate the nose, throat and mucous membrane. High concentrations depress the central nervous system, causing headache, vertigo, poor concentration, sleep and failure of the heart and breathing. Some ketones can cause multiple nerve disorders, inducing "pins and needles" and weakness in the limbs.  The most common signs of inhalation overexposure to ethanol, in animals, include ataxia, incoordination and drowsiness for those surviving narcosis. The narcotic dose for rats, after 2 hours of exposure, is 19260 ppm.  

CHRONIC HEALTH EFFECTS

  Harmful: danger of serious damage to health by prolonged exposure through inhalation.  This material can cause serious damage if one is exposed to it for long periods. It can be assumed that it contains a substance which can produce severe defects. This has been demonstrated via both short- and long-term experimentation.  Results in experiments suggest that this material may cause disorders in the development of the embryo or fetus, even when no signs of poisoning show in the mother.  Limited evidence suggests that repeated or long-term occupational exposure may produce cumulative health effects involving organs or biochemical systems.  Constant or exposure over long periods to mixed hydrocarbons may produce stupor with dizziness, weakness and visual disturbance, weight loss and anemia, and reduced liver and kidney function. Skin exposure may result in drying and cracking and redness of the skin. Chronic exposure to lighter hydrocarbons can cause nerve damage, peripheral neuropathy, bone marrow dysfunction and psychiatric disorders as well as damage the liver and kidneys.  Prolonged exposure to ethanol may cause damage to the liver and cause scarring. It may also worsen damage caused by other agents. Large amounts of ethanol taken in pregnancy may result in "fetal alcohol syndrome", characterized by delay in mental and physical development, learning difficulties, behavioral problems and small head size. A small number of people develop allergic reactions to ethanol, which include eye infections, skin swelling, shortness of breath, and itchy rashes with blisters.  Long term or repeated ingestion exposure of isopropanol may produce incoordination, lethargy and reduced weight gain.  Repeated inhalation exposure to isopropanol may produce narcosis, incoordination and liver degeneration. Animal data show developmental effects only at exposure levels that produce toxic effects in the adult animals. Isopropanol does not cause genetic damage in bacterial or mammalian cell cultures or in animals.  There are inconclusive reports of human sensitisation from skin contact with isopropanol. Chronic alcoholics are more tolerant of systemic isopropanol than are persons who do not consume alcohol; alcoholics have survived as much as 500 ml. of 70% isopropanol.  Continued voluntary drinking of a 2.5% aqueous solution through two successive generations of rats produced no reproductive effects.  NOTE: Commercial isopropanol doers not contain "isopropyl oil". An excess incidence of sinus and laryngeal cancers in isopropanol production workers has been shown to be caused by the byproduct "isopropyl oil". Changes in the production processes now ensure that no byproduct is formed. Production changes include use of dilute sulfuric acid at higher temperatures.  Chronic exposure to benzene may cause headache, fatigue, loss of appetite and lassitude with incipient blood effects including anaemia and blood changes. Benzene is a myelotoxicant known to suppress bone- marrow cell proliferation and to induce haematologic disorders in humans and animals. Signs of benzene-induced aplastic anaemia include suppression of leukocytes (leukopenia), red cells (anaemia), platelets (thrombocytopenia) or all three cell types (pancytopenia). Classic symptoms include weakness, purpura, and haemorrhage. The most significant toxic effect is insidious and often reversible injury to the blood forming tissue. Leukaemia may develop. Occupational exposures have shown a relationship between exposure to benzene and production of myelogenous leukaemia. There may also be a relationship between benzene exposure and the production of lymphoma and multiple myeloma. In chronic exposure, workers exhibit signs of central nervous system lesions and impairment of hearing.Benzene haemotoxicity and leukaemogenicity involve metabolism, growth factor regulation, oxidative stress, DNA damage, cell regulation, and apoptosis. (Yoon et al Environmental Health Perspectives, 111, pp 1411-1420, 2003).  Chronic solvent inhalation exposures may result in nervous system impairment and liver and blood changes. [PATTYS].