Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

JASOL RELEASE (AEROSOL)

NFPA

PRODUCT USE

The use of a quantity of material in an unventilated or confined space may result in
increased exposure and an irritating atmosphere developing.Before starting consider
control of exposure by mechanical ventilation. Application is by spray atomization from a
hand held aerosol pack. Silicone lubricant and release agent.

SYNONYMS

"silicone lubricant release agent"

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

Harmful if swallowed.
Irritating to skin.
Limited evidence of a carcinogenic effect.
Extremely flammable.
Risk of explosion if heated under confinement.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual.  Swallowing of the liquid may cause aspiration into the lungs with the risk of chemical pneumonitis; serious consequences may result. (ICSC13733).  Central nervous system (CNS) depression may include general discomfort, symptoms of giddiness, headache, dizziness, nausea, anaesthetic effects, slowed reaction time, slurred speech and may progress to unconsciousness. Serious poisonings may result in respiratory depression and may be fatal.  

EYE

  There is some evidence that material may produce eye irritation in some persons and produce eye damage 24 hours or more after instillation. Moderate inflammation may be expected with redness; conjunctivitis may occur with prolonged exposure.  Not considered to be a risk because of the extreme volatility of the gas.  

SKIN

  The material may cause moderate inflammation of the skin either following direct contact or after a delay of some time. Repeated exposure can cause contact dermatitis which is characterized by redness, swelling and blistering.  Skin contact with the material may damage the health of the individual; systemic effects may result following absorption.  Spray mist may produce discomfort.  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  

INHALED

  Inhalation may produce health damage*.  Inhalation of vapours may cause drowsiness and dizziness. This may be accompanied by narcosis, reduced alertness, loss of reflexes, lack of coordination and vertigo.  Inhalation of aerosols (mists, fumes), generated by the material during the course of normal handling, may be damaging to the health of the individual.  There is some evidence to suggest that the material can cause respiratory irritation in some persons. The body's response to such irritation can cause further lung damage.  Inhalation hazard is increased at higher temperatures.  Inhaling high concentrations of mixed hydrocarbons can cause narcosis, with nausea, vomiting and lightheadedness. Low molecular weight (C2-C12) hydrocarbons can irritate mucous membranes and cause incoordination, giddiness, nausea, vertigo, confusion, headache, appetite loss, drowsiness, tremors and stupor. Massive exposures can lead to severe central nervous system depression, deep coma and death. Convulsions can occur due to brain irritation and/or lack of oxygen. Permanent scarring may occur, with epileptic seizures and brain bleeds occurring months after exposure. Respiratory system effects include inflammation of the lungs with edema and bleeding. Lighter species mainly cause kidney and nerve damage; the heavier paraffins and olefins are especially irritant to the respiratory system. Alkenes produce pulmonary edema at high concentrations. Liquid paraffins may produce sensation loss and depressant actions leading to weakness, dizziness, slow and shallow respiration, unconsciousness, convulsions and death. C5-7 paraffins may also produce multiple nerve damage. Aromatic hydrocarbons accumulate in lipid rich tissues (typically the brain, spinal cord and peripheral nerves) and may produce functional impairment manifested by nonspecific symptoms such as nausea, weakness,  fatigue, vertigo; severe exposures may produce inebriation or unconsciousness. Many of the petroleum hydrocarbons can sensitize the heart and may cause ventricular fibrillation,  leading to death.  Central nervous system (CNS) depression may include general discomfort, symptoms of giddiness, headache, dizziness, nausea, anaesthetic effects, slowed reaction time, slurred speech and may progress to unconsciousness. Serious poisonings may result in respiratory depression and may be fatal.  Material is highly volatile and may quickly form a concentrated atmosphere in confined or unventilated areas. Vapor is heavier than air and may displace and replace air in breathing zone, acting as a simple asphyxiant. This may happen with little warning of overexposure.  Inhalation of high concentrations of gas/vapor causes lung irritation with coughing and nausea, central nervous depression with headache and dizziness, slowing of reflexes, fatigue and inco-ordination.  WARNING: Intentional misuse by concentrating/inhaling contents may be lethal.  Inhalation exposure may cause susceptible individuals to show change in heart beat rhythm i.e. cardiac arrhythmia. Exposures must be terminated.  At high concentrations most of the absorbed methylene chloride (dichloromethane) is exhaled unchanged; the remainder is metabolised to carbon monoxide, carbon dioxide and inorganic chloride. Inhalation may produce fatigue, weakness, sleepiness, light-  headedness, chills, nausea, diarrhoea and abdominal pain. The lowest published lethal dose is 20,000 ppm for 20 hours. The body metabolises methylene chloride to carbon monoxide and adds to the body burden of carboxyhaemoglobin (COHb) contributed by other sources. The increase in COHb is related to the magnitude of vapour exposure and duration. Serious poisoning can occur without raised COHb concentrations, although these raised concentrations may persist for several hours. Central nervous system (CNS) effects are thought to be due to methylene chloride itself or methylene chloride in combination with other sources of COHb, rather than the COHb metabolite. The raised COHb concentrations are not usually expected to produce adverse effects in healthy individuals but may be cause for concern in individuals with cardiovascular disease. Encephalopathy (brain injury) has been reported after repeated exposure. Angina, myocardial infarction, cardiac arrhythmias and cardiac arrest have also been reported, although the cardiovascular system is not generally a target for methylene chloride toxicity. Hypotension, shock and metabolic acidosis may also occur as a result of overexposure. Respiratory failure may develop, secondary to CNS depression, in severe cases.  

CHRONIC HEALTH EFFECTS

  There has been concern that this material can cause cancer or mutations, but there is not enough data to make an assessment.  Limited evidence suggests that repeated or long-term occupational exposure may produce cumulative health effects involving organs or biochemical systems.  Principal route of occupational exposure to the gas is by inhalation.  Constant or exposure over long periods to mixed hydrocarbons may produce stupor with dizziness, weakness and visual disturbance, weight loss and anemia, and reduced liver and kidney function. Skin exposure may result in drying and cracking and redness of the skin. Chronic exposure to lighter hydrocarbons can cause nerve damage, peripheral neuropathy, bone marrow dysfunction and psychiatric disorders as well as damage the liver and kidneys.  Dichloromethane exposures cause liver and kidney damage in animals and this justifies consideration before exposing persons with a history of impaired liver function and/or renal disorders.