JOHNSON BAYGON LOW IRRITANT SURFACE SPRAY FORMULA
Flammability | 1 | |
Toxicity | 3 | |
Body Contact | 2 | |
Reactivity | 1 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Application is by spray atomization from a hand held aerosol pack.
insecticide
Harmful by inhalation.
Toxic if swallowed.
Extremely flammable.
Harmful to aquatic organisms, may cause long- term adverse effects in the
aquatic environment.
Risk of explosion if heated under confinement.
Not normally a hazard due to physical form of product. Considered an unlikely route of entry in commercial/industrial environments. Toxic effects may result from the accidental ingestion of the material; animal experiments indicate that ingestion of less than 40 gram may be fatal or may produce serious damage to the health of the individual. Swallowing of the liquid may cause aspiration into the lungs with the risk of chemical pneumonitis; serious consequences may result. (ICSC13733). Ingestion may produce nausea, vomiting, depressed appetite, abdominal cramps,and diarrhea. Constant or exposure over long periods to mixed hydrocarbons may produce stupor with dizziness, weakness and visual disturbance, weight loss and anemia, and reduced liver and kidney function. Skin exposure may result in drying and cracking and redness of the skin. Chronic exposure to lighter hydrocarbons can cause nerve damage, peripheral neuropathy, bone marrow dysfunction and psychiatric disorders as well as damage the liver and kidneys. Central nervous system (CNS) depression may include general discomfort, symptoms of giddiness, headache, dizziness, nausea, anaesthetic effects, slowed reaction time, slurred speech and may progress to unconsciousness. Serious poisonings may result in respiratory depression and may be fatal.
Although the material is not thought to be an irritant, direct contact with the eye may produce transient discomfort characterized by tearing or conjunctival redness (as with windburn).
Spray mist may produce discomfort. Skin contact is not thought to produce harmful health effects (as classified using animal models). Systemic harm, however, has been identified following exposure of animals by at least one other route and the material may still produce health damage following entry through wounds, lesions or abrasions. Good hygiene practice requires that exposure be kept to a minimum and that suitable gloves be used in an occupational setting. Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.
Inhalation of aerosols (mists, fumes), generated by the material during the course of normal handling, may be harmful. Inhalation of toxic gases may cause: · Central Nervous System effects including depression, headache, confusion, dizziness, stupor, coma and seizures; · respiratory: acute lung swellings, shortness of breath, wheezing, rapid breathing, other symptoms and respiratory arrest; · heart: collapse, irregular heartbeats and cardiac arrest; · gastrointestinal: irritation, ulcers, nausea and vomiting (may be bloody), and abdominal pain. The material is not thought to produce respiratory irritation (as classified using animal models). Nevertheless inhalation of the material, especially for prolonged periods, may produce respiratory discomfort and occasionally, distress. Inhalation of high concentrations of gas/vapor causes lung irritation with coughing and nausea, central nervous depression with headache and dizziness, slowing of reflexes, fatigue and inco-ordination. Central nervous system (CNS) depression may include general discomfort, symptoms of giddiness, headache, dizziness, nausea, anaesthetic effects, slowed reaction time, slurred speech and may progress to unconsciousness. Serious poisonings may result in respiratory depression and may be fatal. WARNING: Intentional misuse by concentrating/inhaling contents may be lethal. Inhalation of vapours may cause drowsiness and dizziness. This may be accompanied by narcosis, reduced alertness, loss of reflexes, lack of coordination and vertigo. Poisoning due to cholinesterase inhibitors causes symptoms such as increased blood flow to the nose, watery discharge, chest discomfort, shortness of breath and wheezing. Other symptoms include increased production of tears, nausea and vomiting, diarrhea, stomach pain, involuntary passing of urine and stools, chest pain, breathing difficulty, low blood pressure, irregular heartbeat, loss of reflexes, twitching, visual disturbances, altered pupil size, convulsions, lung congestion, coma and heart failure. Nervous system effects include inco-ordination, slurred speech, tremors of the tongue and eyelids, and paralysis of the limbs and muscles of breathing, which can cause death, although death is also seen due to cardiac arrest. Symptoms of carbamate poisoning are similar to that of organophosphate poisoning, however, recover from carbamate poisoning is quicker and generally less likely to be cause death. This material, like natural pyrethrins, may cause central stimulation with nausea, vomiting, stomach upset, diarrhea, hypersensitivity, inco-ordination, tremors, muscle paralysis, convulsion, coma and respiratory failure. Type II compounds cause a "Type II syndrome" characterized by irregular jerky movements, increased saliva production without tears, upper abdominal pain, nausea and vomiting, headache, dizziness, loss of appetite, tiredness, chest tightness, blurred vision, "pins and needles", palpitations, coarse muscle jerks in limbs and altered consciousness. Convulsions can occur in severe cases with flexed arms, extended legs (spastic posture) and unconsciousness. Recovery may take weeks.
There is some evidence that inhaling this product is more likely to cause a sensitization reaction in some persons compared to the general population.
Principal route of occupational exposure to the gas is by inhalation. Limited evidence suggests that repeated or long-term occupational exposure may produce cumulative health effects involving organs or biochemical systems. Repeated or prolonged exposures to cholinesterase inhibitors produce symptoms similar to acute effects. In addition workers exposed repeatedly to these substances may exhibit impaired memory and loss of concentration, severe depression and acute psychosis, irritability, confusion, apathy, emotional liability, speech difficulties, headache, spatial disorientation, delayed reaction times, sleepwalking, drowsiness or insomnia. An influenza-like condition with nausea, weakness, anorexia and malaise has been described. There is a growing body of evidence from epidemiological studies and from experimental laboratory studies that short-term exposure to some cholinesterase-inhibiting insecticides may produce behavioral or neuro- chemical changes lasting for days or months, presumably outlasting the cholinesterase inhibition. Although the number of adverse effects following humans poisonings subside, there are still effects in some workers months after cholinesterase activity returns to normal. These long-lasting effects include blurred vision, headache, weakness, and anorexia. The neurochemistry of animals exposed to chlorpyrifos or fenthion is reported to be altered permanently after a single exposure. These effects may be more severe in developing animals where both acetyl- and butyrylcholinesterase may play an integral part in the development of the nervous system. Padilla S., The Neurotoxicity of Cholinesterase-Inhibiting Insecticides: Past and Present Evidence Demonstrating Persistent Effects. Inhalation Toxicology 7:903-907, 1995. Constant or exposure over long periods to mixed hydrocarbons may produce stupor with dizziness, weakness and visual disturbance, weight loss and anemia, and reduced liver and kidney function. Skin exposure may result in drying and cracking and redness of the skin. Chronic exposure to lighter hydrocarbons can cause nerve damage, peripheral neuropathy, bone marrow dysfunction and psychiatric disorders as well as damage the liver and kidneys.