Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

VIKING 2000

NFPA

PRODUCT USE

Non- flammable adhesive for bonding rubber to rubber, metal, cement, wood and many other
materials.

SYNONYMS

"Cold Vulcanising Cement"

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

May cause CANCER.
Possible risk of irreversible effects.
Irritating to eyes and skin.
Vapors may cause dizziness or suffocation.
Harmful to aquatic organisms, may cause long- term adverse effects in the
aquatic environment.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Accidental ingestion of the material may be damaging to the health of the individual.  Ingestion may result in nausea, abdominal irritation, pain and vomiting.  

EYE

  This material can cause eye irritation and damage in some persons.  The material may produce moderate eye irritation leading to inflammation. Repeated or prolonged exposure to irritants may produce conjunctivitis.  

SKIN

  This material can cause inflammation of the skin oncontact in some persons.  Skin contact with the material may damage the health of the individual; systemic effects may result following absorption.  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  The material may cause severe skin irritation after prolonged or repeated exposure and may produce on contact skin redness, swelling, the production of vesicles, scaling and thickening of the skin. Repeated exposures may produce severe ulceration.  

INHALED

  Inhalation may produce health damage*.  Inhalation of vapors or aerosols (mists, fumes), generated by the material during the course of normal handling, may be damaging to the health of the individual.  There is some evidence to suggest that the material can cause respiratory irritation in some persons. The body's response to such irritation can cause further lung damage.  Anesthetics and narcotic effects (with dulling of senses and odor fatigue) are a consequence of exposure to chlorinated solvents.  Individual response varies widely; odor may not be considered objectionable at levels which quickly induce central nervous system effects. High vapor concentrations may give a feeling of euphoria. This may result in reduced responses, followed by rapid onset of unconsciousness, possible respiratory arrest and death.  Systemic effects of trichloroethylene (TCE) exposure involve the central nervous system and produce headache, light-headedness, dizziness, ataxia, euphoria, confusion, drowsiness, and coma. Other adverse findings include nausea, vomiting, hypotension, bradycardia or tachycardia and hepatitis. Deaths may occur from ventricular arrhythmias as a result of sensitization of the myocardium to adrenaline or other catecholamines. Recovery from narcotic effects is usually rapid following cessation of exposure.  Trigeminal nerve impairment and peripheral neuropathy have been reported following TCE exposure.  Evidence of acute human toxicity comes mainly from the use of TCE as an anesthetic. Tachypnea and ventricular arrhythmias are experienced at inhaled concentrations exceeding 15000 ppm. Systemic toxicity is low following anesthesia. Occasional hepatotoxicity (liver dysfunction) has been reported; this is probably due to the breakdown of TCE to dichloroacetylene and phosgene by soda-lime present in some anesthetic devices.  The effects of TCE appear to be enhanced in some individuals by simultaneous exposure to caffeine, ethanol and other drugs. "Degreaser's Flush" describes a reddening of facial, neck and back skin and is seen by certain individuals after exposure to TCE.  

CHRONIC HEALTH EFFECTS

  There is ample evidence that this material can be regarded as being able to cause cancer in humans based on experiments and other information.  There is some evidence that human exposure to the material may result in developmental toxicity. This evidence is based on animal studies where effects have been observed in the absence of marked maternal toxicity, or at around the same dose levels as other toxic effects but which are not secondary non-specific consequences of the other toxic effects.  Exposure to the material may result in a possible risk of irreversible effects. The material may produce mutagenic effects in man. This concern is raised, generally, on the basis ofappropriate studies using mammalian somatic cells in vivo. Such findings are often supported by positive results from in vitro mutagenicity studies.