UNITED CATALYSTS CONTAINER DRI
Flammability | 0 | |
Toxicity | 2 | |
Body Contact | 2 | |
Reactivity | 0 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Packaged desiccant / moisture absorbant.
"absorbent moisture absorber desiccant dessicant", "Dry (misspelling)", "bentonite
calcium chloride mixture"
Harmful if swallowed.
Irritating to eyes.
Harmful: danger of serious damage to health by prolonged exposure through
inhalation.
Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual. Considered an unlikely route of entry in commercial/industrial environments. Ingestion may result in nausea, abdominal irritation, pain and diarrhea.
This material can cause eye irritation and damage in some persons. The dust may produce eye discomfort and abrasive eye inflammation. The material may produce severe irritation to the eye causing pronounced inflammation. Repeated or prolonged exposure to irritants may produce conjunctivitis. The material may produce severe irritation to the eye causing pronounced inflammation. Repeated or prolonged exposure to irritants may produce conjunctivitis.
The material is not thought to produce adverse health effects or skin irritation following contact (as classified using animal models). Nevertheless, good hygiene practice requires that exposure be kept to a minimum and that suitable gloves be used in an occupational setting. Solution of material in moisture on the skin, or perspiration, mayincrease irritant effects. The material may cause skin irritation after prolonged or repeated exposure and may produce on contact skin redness, swelling, the production of vesicles, scaling and thickening of the skin.
The material is not thought to produce adverse health effects or irritation of the respiratory tract (as classified using animal models). Nevertheless, good hygiene practice requires that exposure be kept to a minimum and that suitable control measures be used in an occupational setting. Persons with impaired respiratory function, airway diseases and conditions such as emphysema or chronic bronchitis, may incur further disability if excessive concentrations of particulate are inhaled. Effects on lungs are significantly enhanced in the presence of respirableparticles.
Principal routes of exposure are by accidental skin and eye contact andinhalation of generated dusts. Chronic dust inhalation has been associated with lung disease. (Source: NIOSHTIC). Symptoms are those of nodular fibrosis and respiratory impairment is characterised by obstruction and restriction of lung function. (Source: Occupational Diseases) Long term exposure to high dust concentrations may cause changes in lung function i.e. pneumoconiosis; caused by particles less than 0.5 micron penetrating and remaining in the lung. Prime symptom is breathlessness; lung shadows show on X-ray. Repeated exposures, in an occupational setting, to high levels of fine- divided dusts may produce a condition known as pneumoconiosis which is the lodgement of any inhaled dusts in the lung irrespective of the effect. This is particularly true when a significant number of particles less than 0.5 microns (1/50,000 inch), are present. Lung shadows are seen in the X-ray. Symptoms of pneumoconiosis may include a progressive dry cough, shortness of breath on exertion, increased chest expansion, weakness and weight loss. As the disease progresses the cough produces a stringy mucous, vital capacity decreases further and shortness of breath becomes more severe. Pneumoconiosis is the accumulation of dusts in the lungs and the tissue reaction in its presence. It is further classified as being of noncollagenous or collagenous types. Noncollagenous pneumoconiosis, the benign form, is identified by minimal stromal reaction, consists mainly of reticulin fibres, an intact alveolar architecture and is potentially reversible. The clay portion of this mixture may contain a small amount of crystalline silica (quartz). Crystalline silicas activate the inflammatory response of white blood cells after they injure the lung epithelium. Chronic exposure to crystalline silicas reduce lung capacity and predispose to chest infections. A large part of the crystals accumulates in the lungs. Silicosis can occur, a condition where irreversible scarring of the lung occurs. Symptoms do not appear until months to years after exposure. Smoking increases this risk. Most simple cases of silicosis do not produce symptoms, but they can progress and eventually cause a tuberculosis-like syndrome which can be fatal. When silicosis is advanced, there is an increased risk of lung cancer and lymphoma. Laws in some areas require those exposed to silica to be under health surveillance.