UOP KG-1-5B UNIONFINING CATALYST
Flammability | 0 | |
Toxicity | 0 | |
Body Contact | 2 | |
Reactivity | 0 | |
Chronic | 4 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Catalyst. Used according to manufacturer' s directions.
catalyst, "KG 1 5B"
Danger of cumulative effects.
May cause CANCER by inhalation.
May cause SENSITIZATION by skin contact.
Harmful: danger of serious damage to health by prolonged exposure through
inhalation and if swallowed.
Irritating to eyes and respiratory system.
May cause long- term adverse effects in the aquatic environment.
The material has NOT been classified as "harmful by ingestion". This is because of the lack of corroborating animal or human evidence. The material may still be damaging to the health of the individual, following ingestion, especially where pre-existing organ (e.g. liver, kidney) damage is evident. Present definitions of harmful or toxic substances are generally based on doses producing mortality (death) rather than those producing morbidity (disease, ill-health). Gastrointestinal tract discomfort may produce nausea and vomiting. In an occupational setting however, unintentional ingestion is not thought to be cause for concern.
This material can cause eye irritation and damage in some persons.
The material is not thought to produce adverse health effects or skin irritation following contact (as classified using animal models). Nevertheless, good hygiene practice requires that exposure be kept to a minimum and that suitable gloves be used in an occupational setting. Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.
The material can cause respiratory irritation in some persons. The body's response to such irritation can cause further lung damage. Persons with impaired respiratory function, airway diseases and conditions such as emphysema or chronic bronchitis, may incur further disability if excessive concentrations of particulate are inhaled.
Repeated or long-term occupational exposure is likely to produce cumulative health effects involving organs or biochemical systems. There is some evidence that inhaling this product is more likely to cause a sensitization reaction in some persons compared to the general population. Skin contact with the material is more likely to cause a sensitization reaction in some persons compared to the general population. There is sufficient evidence to suggest that this materialdirectly causes cancer in humans. Exposure to large doses of Aluminum has been connected with the degenerative brain disease Alzheimer's Disease. High levels of molybdenum can cause joint problems in the hands and feet with pain and lameness. Molybdenum compounds can also cause liver changes with elevated levels of enzymes and cause over-activity of the thyroid gland. A generalized feeling of unwellness can occur, with tiredness, weakness, diarrhea, loss of appetite and weight. Molybdenum has been associated with cancers of the airways, but on the other hand, a low intake of molybdenum may cause an increased risk of developing esophageal cancer. Repeated exposures, in an occupational setting, to high levels of fine- divided dusts may produce a condition known as pneumoconiosis which is the lodgement of any inhaled dusts in the lung irrespective of the effect. This is particularly true when a significant number of particles less than 0.5 microns (1/50,000 inch), are present. Lung shadows are seen in the X-ray. Symptoms of pneumoconiosis may include a progressive dry cough, shortness of breath on exertion, increased chest expansion, weakness and weight loss. As the disease progresses the cough produces a stringy mucous, vital capacity decreases further and shortness of breath becomes more severe. Pneumoconiosis is the accumulation of dusts in the lungs and the tissue reaction in its presence. It is further classified as being of noncollagenous or collagenous types. Noncollagenous pneumoconiosis, the benign form, is identified by minimal stromal reaction, consists mainly of reticulin fibres, an intact alveolar architecture and is potentially reversible.