UNIMIN FIRECLAY
Flammability | 0 | |
Toxicity | 2 | |
Body Contact | 2 | |
Reactivity | 0 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Fireclay.
"Fire clay aluminium silicates"
Harmful: danger of serious damage to health by prolonged exposure through
inhalation.
Although ingestion is not thought to produce harmful effects, the material may still be damaging to the health of the individual following ingestion, especially where pre- existing organ (e.g. liver, kidney) damage is evident. Present definitions of harmful or toxic substances are generally based on doses producing mortality (death) rather than those producing morbidity (disease, ill-health). Gastrointestinal tract discomfort may produce nausea and vomiting. In an occupational setting however, ingestion of insignificant quantities is not thought to be cause for concern. Considered an unlikely route of entry in commercial/industrial environments. Ingestion may result in nausea, abdominal irritation, pain and vomiting. Considered to be non toxic.
Although the material is not thought to be an irritant, direct contact with the eye may produce transient discomfort characterized by tearing or conjunctival redness (as with windburn). The dust may produce eye discomfort and abrasive eye inflammation.
The material is not thought to produce adverse health effects or skin irritation following contact (as classified using animal models). Nevertheless, good hygiene practice requires that exposure be kept to a minimum and that suitable gloves be used in an occupational setting.
The material is not thought to produce adverse health effects or irritation of the respiratory tract (as classified using animal models). Nevertheless, good hygiene practice requires that exposure be kept to a minimum and that suitable control measures be used in an occupational setting. Not normally a hazard due to non-volatile nature of product. Persons with impaired respiratory function, airway diseases and conditions such as emphysema or chronic bronchitis, may incur further disability if excessive concentrations of particulate are inhaled. Effects on lungs are significantly enhanced in the presence of respirableparticles. Acute silicosis occurs under conditions of extremely high silica dust exposure particularly when the particle size of the dust is small. The disease is rapidly progressive and spreads widely through the lungs within months of the initial exposure and causing deaths within 1 to 2 years.
Principal routes of exposure are usually by inhalation of generated dust and skin contact with the material. Inhalation of dusts containing crystalline silicas may lead to silicosis. Effects are cumulative, with scarring, impairment of breathing, emphysema, and restriction and obstruction of lung function. Chronic symptoms include decreased lung capacity and chest infections. Scarring often does not appear until after many months of exposure, and smoking increases the risk. Silicosis can progress even when exposure is removed. It has been claimed that silicosis can increase the risk of cancer of the lung and bronchi developing. Some jurisdictions require health surveillance be conducted on workers occupationally exposed to crystalline silica.