Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

WEAR PROTECT WPS 105 PART B

NFPA

PRODUCT USE

Curing agent used as part of an abrasion resistant wear compound.

SYNONYMS

"curing agent"

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

Causes burns.
Risk of serious damage to eyes.
May cause SENSITIZATION by skin contact.
May cause harm to the unborn child.
Possible risk of irreversible effects.
Harmful by inhalation, in contact with skin and if swallowed.
Harmful to aquatic organisms, may cause long- term adverse effects in the
aquatic environment.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual.  The material can produce chemical burns within the oral cavity and gastrointestinal tract following ingestion.  Strong evidence exists that the substance may cause irreversible but non-lethal mutagenic effects following a single exposure.  

EYE

  The material can produce chemical burns to the eye following direct contact. Vapors or mists may be extremely irritating.  If applied to the eyes, this material causes severe eye damage.  

SKIN

  Skin contact with the material may be harmful; systemic effects may resultfollowing absorption.  The material can produce chemical burns following direct contactwith the skin.  Open cuts, abraded or irritated skin should not be exposed to this material.  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  

INHALED

  If inhaled, this material can irritate the throat andlungs of some persons.  The inhalation of small particles of metal oxide results in sudden thirst, a sweet, metallic our foul taste, throat irritation, cough, dry mucous membranes, tiredness and general unwellness. Headache, nausea and vomiting, fever or chills, restlessness, sweating, diarrhea, excessive urination and prostration may also occur. After exposure is removed, recovery occurs within 24-36 hours.  

CHRONIC HEALTH EFFECTS

  Repeated or prolonged exposure to corrosives may result in the erosion of teeth, inflammatory and ulcerative changes in the mouth and necrosis (rarely) of the jaw. Bronchial irritation, with cough, and frequent attacks of bronchial pneumonia may ensue. Gastrointestinal disturbances may also occur. Chronic exposures may result in dermatitis and/or conjunctivitis.  Skin contact with the material is more likely to cause a sensitization reaction in some persons compared to the general population.  Ample evidence exists, from results in experimentation, that developmental disorders are directly caused by human exposure to the material.  Exposure to the material may result in a possible risk of irreversible effects. The material may produce mutagenic effects in man. This concern is raised, generally, on the basis ofappropriate studies using mammalian somatic cells in vivo. Such findings are often supported by positive results from in vitro mutagenicity studies.  Limited evidence suggests that repeated or long-term occupational exposure may produce cumulative health effects involving organs or biochemical systems.  There is some evidence that inhaling this product is more likely to cause a sensitization reaction in some persons compared to the general population.  Solid phenol is highly toxic via ingestion, inhalation and skin contact.  Chronic phenol poisoning is very rarely reported, but symptoms include vomiting, difficulty in swallowing, diarrhoea, lack of appetite, headache, fainting, dizziness, dark urine, mental disturbances, and possibly skin rash. Death due to liver and kidney damage may occur.  Repeated exposure of animals to phenol vapour at concentrations ranging from 26 to 52 ppm has produced respiratory, cardiovascular, hepatic, renal and neurologic toxicity.  Administration of phenol in the drinking water of mice (2500 ppm for 103 weeks) produced an increased incidence of leukemia and lymphomas.  Phenol has been studied in initiation/promotion protocols with a number of polycyclic hydrocarbons and has been shown to have promoting activity in the two-stage skin model.  repeated exposure may cause sensitization and/or allergic reactions.  Sensitization may give severe responses to very low levels of exposure, i.e. hypersensitivity. Sensitized persons should not be allowed to work in situations where exposure may occur.  Inhalation of epoxy resin amine hardeners (including polyamines and amine adducts) and may produce bronchospasm and coughing episodes lasting several days after cessation of the exposure. Even faint traces of these vapors may trigger an intense reaction in individuals showing "amine asthma". The literature records several instances of systemic intoxications following the use of amines in epoxy resin systems.