WHITE CEMENT
Flammability | 0 | |
Toxicity | 2 | |
Body Contact | 3 | |
Reactivity | 0 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
As building material.
Causes burns.
Risk of serious damage to eyes.
The material can produce chemical burns within the oral cavity and gastrointestinal tract following ingestion.
If applied to the eyes, this material causes severe eye damage. The material can produce chemical burns to the eye following direct contact. Vapors or mists may be extremely irritating.
The material can produce chemical burns following direct contactwith the skin. Handling wet cement can cause dermatitis. Cement when wet is quite alkaline and this alkali action on the skin contributes strongly to cement contact dermatitis since it may cause drying and defatting of the skin which is followed by hardening, cracking, lesions developing, possible infections of lesions and penetration by soluble salts. Cement contact dermatitis (CCD) may occur when contact shows an allergic response, which may progress to sensitization. Sensitization is due to soluble chromates (chromate compounds) present in trace amounts in some cements, cement products. Soluble chromates readily penetrate intact skin. Cement dermatitis can be characterized by fissures, eczematous rash, dystrophic nails, and dry skin; acute contact with highly alkaline mixtures may cause localized necrosis.
If inhaled, this material can irritate the throat andlungs of some persons. Inhalation of dusts, generated by the material during the course of normal handling, may be damaging to the health of the individual. Effects on lungs are significantly enhanced in the presence of respirableparticles.
Repeated exposures, in an occupational setting, to high levels of fine- divided dusts may produce a condition known as pneumoconiosis which is the lodgement of any inhaled dusts in the lung irrespective of the effect. This is particularly true when a significant number of particles less than 0.5 microns (1/50,000 inch), are present. Lung shadows are seen in the X-ray. Symptoms of pneumoconiosis may include a progressive dry cough, shortness of breath on exertion, increased chest expansion, weakness and weight loss. As the disease progresses the cough produces a stringy mucous, vital capacity decreases further and shortness of breath becomes more severe. Pneumoconiosis is the accumulation of dusts in the lungs and the tissue reaction in its presence. It is further classified as being of noncollagenous or collagenous types. Noncollagenous pneumoconiosis, the benign form, is identified by minimal stromal reaction, consists mainly of reticulin fibres, an intact alveolar architecture and is potentially reversible. There is some evidence that inhaling this product is more likely to cause a sensitization reaction in some persons compared to the general population. There is limited evidence that, skin contact with this product is more likely to cause a sensitization reaction in some persons compared to the general population. Respiratory sensitization may result in allergic/asthma like responses; from coughing and minor breathing difficulties to bronchitis with wheezing, gasping.