Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

WATTYL ESTAPOL GLOSS AEROSOL

NFPA

PRODUCT USE

The use of a quantity of material in an unventilated or confined space may result in
increased exposure and an irritating atmosphere developing.Before starting consider
control of exposure by mechanical ventilation. Application is by spray atomization from a
hand held aerosol pack. Spraying of small areas, for repair, touch up in the home or
industry.

SYNONYMS

"Aerosol paint enamel", "touch up spray can"

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

Limited evidence of a carcinogenic effect.
Possible risk of harm to the unborn child.
Harmful: danger of serious damage to health by prolonged exposure through
inhalation.
Harmful by inhalation and if swallowed.
Irritating to eyes, respiratory system and skin.
Extremely flammable.
Vapors may cause dizziness or suffocation.
Toxic to aquatic organisms, may cause long- term adverse effects in the aquatic
environment.
Risk of explosion if heated under confinement.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual.  Not normally a hazard due to physical form of product.  Considered an unlikely route of entry in commercial/industrial environments.  Considered an unlikely route of entry in commercial/industrial environments. The liquid may produce gastrointestinal discomfort and may be harmful if swallowed. Ingestion may result in nausea, pain and vomiting. Vomit entering the lungs by aspiration may cause potentially lethal chemical pneumonitis.  Central nervous system (CNS) depression may include general discomfort, symptoms of giddiness, headache, dizziness, nausea, anaesthetic effects, slowed reaction time, slurred speech and may progress to unconsciousness. Serious poisonings may result in respiratory depression and may be fatal.  

EYE

  There is evidence that material may produce eye irritation in some persons and produce eye damage 24 hours or more after instillation. Severe inflammation may be expected with pain. There may be damage to the cornea. Unless treatment is prompt and adequate there may be permanent loss of vision. Conjunctivitis can occur following repeated exposure.  Not considered to be a risk because of the extreme volatility of the gas.  The vapour when concentrated has pronounced eye irritation effects and this gives some warning of high vapour concentrations. If eye irritation occurs seek to reduce exposure with available control measures, or evacuate area.  Direct eye contact with petroleum hydrocarbons can be painful, and the corneal epithelium may be temporarily damaged. Aromatic species can cause irritation and excessive tear secretion.  

SKIN

  Skin contact with the material may damage the health of the individual; systemic effects may result following absorption.  Spray mist may produce discomfort.  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  The material may cause moderate inflammation of the skin either following direct contact or after a delay of some time. Repeated exposure can cause contact dermatitis which is characterized by redness, swelling and blistering.  Aromatic hydrocarbons may produce sensitivity and redness of the skin. They are not likely to be absorbed into the body through the skin but branched species are more likely to.  

INHALED

  Inhalation of aerosols (mists, fumes), generated by the material during the course of normal handling, may be harmful.  The material can cause respiratory irritation in some persons. The body's response to such irritation can cause further lung damage.  Inhalation hazard is increased at higher temperatures.  Inhalation of high concentrations of gas/vapor causes lung irritation with coughing and nausea, central nervous depression with headache and dizziness, slowing of reflexes, fatigue and inco-ordination.  If exposure to highly concentrated solvent atmosphere is prolonged this may lead to narcosis, unconsciousness, even coma and possible death.  Material is highly volatile and may quickly form a concentrated atmosphere in confined or unventilated areas. Vapor is heavier than air and may displace and replace air in breathing zone, acting as a simple asphyxiant. This may happen with little warning of overexposure.  WARNING: Intentional misuse by concentrating/inhaling contents may be lethal.  Inhalation exposure may cause susceptible individuals to show change in heart beat rhythm i.e. cardiac arrhythmia. Exposures must be terminated.  Acute intoxication by halogenated aliphatic hydrocarbons appears to take place over two stages. Signs of a reversible narcosis are evident in the first stage and in the second stage signs of injury to organs may become evident, a single organ alone is (almost) never involved.  At high concentrations most of the absorbed methylene chloride (dichloromethane) is exhaled unchanged; the remainder is metabolised to carbon monoxide, carbon dioxide and inorganic chloride. Inhalation may produce fatigue, weakness, sleepiness, light-  headedness, chills, nausea, diarrhoea and abdominal pain. The lowest published lethal dose is 20,000 ppm for 20 hours. The body metabolises methylene chloride to carbon monoxide and adds to the body burden of carboxyhaemoglobin (COHb) contributed by other sources. The increase in COHb is related to the magnitude of vapour exposure and duration. Serious poisoning can occur without raised COHb concentrations, although these raised concentrations may persist for several hours. Central nervous system (CNS) effects are thought to be due to methylene chloride itself or methylene chloride in combination with other sources of COHb, rather than the COHb metabolite. The raised COHb concentrations are not usually expected to produce adverse effects in healthy individuals but may be cause for concern in individuals with cardiovascular disease. Encephalopathy (brain injury) has been reported after repeated exposure. Angina, myocardial infarction, cardiac arrhythmias and cardiac arrest have also been reported, although the cardiovascular system is not generally a target for methylene chloride toxicity. Hypotension, shock and metabolic acidosis may also occur as a result of overexposure. Respiratory failure may develop, secondary to CNS depression, in severe cases.  

CHRONIC HEALTH EFFECTS

  Harmful: danger of serious damage to health by prolonged exposure through inhalation.  Harmful: danger of serious damage to health by prolonged exposure through inhalation.  This material can cause serious damage if one is exposed to it for long periods. It can be assumed that it contains a substance which can produce severe defects. This has been demonstrated via both short- and long-term experimentation.  Results in experiments suggest that this material may cause disorders in the development of the embryo or fetus, even when no signs of poisoning show in the mother.  Limited evidence suggests that repeated or long-term occupational exposure may produce cumulative health effects involving organs or biochemical systems.  There is some evidence to provide a presumption that human exposure to the material may result in impaired fertility on the basis of: some evidence in animal studies of impaired fertility in the absence of toxic effects, or evidence of impaired fertility occurring at around the same dose levels as other toxic effects but which is not a secondary non-  specific consequence of other toxic effects.  Principal route of occupational exposure to the gas is by inhalation.  Dichloromethane exposures cause liver and kidney damage in animals and this justifies consideration before exposing persons with a history of impaired liver function and/or renal disorders.