WATTYL AP 611 UNDERBODY PUTTY
Flammability | 3 | |
Toxicity | 2 | |
Body Contact | 2 | |
Reactivity | 1 | |
Chronic | 3 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Used according to manufacturer' s directions. The use of a quantity of material in an
unventilated or confined space may result in increased exposure and an irritating
atmosphere developing.
Harmful by inhalation.
HARMFUL - May cause lung damage if swallowed.
Irritating to eyes, respiratory system and skin.
Highly flammable.
Vapors may cause dizziness or suffocation.
Toxic to aquatic organisms, may cause long- term adverse effects in the aquatic
environment.
Accidental ingestion of the material may be damaging to the health of the individual. Swallowing of the liquid may cause aspiration into the lungs with the risk of chemical pneumonitis; serious consequences may result. (ICSC13733). Alkenes are generally of low toxicity but they are consideredaspiration hazards. Isoparaffinic hydrocarbons cause temporary lethargy, weakness,inco-ordination and diarrhea.
There is some evidence to suggest that this material can causeeye irritation and damage in some persons. Workers exposed to fumes of blown bitumens developed keratoconjunctivitis. Direct eye contact with petroleum hydrocarbons can be painful, and the corneal epithelium may be temporarily damaged. Aromatic species can cause irritation and excessive tear secretion.
Skin contact with the material may damage the health of the individual; systemic effects may result following absorption. There is some evidence to suggest that this material can cause inflammation of the skin on contact in some persons. Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected. Toxic effects may result from skin absorption. Exposure limits with "skin" notation indicate that vapor and liquid may be absorbed through intact skin. Absorption by skin may readily exceed vapor inhalation exposure. Symptoms for skin absorption are the same as for inhalation. Contact with eyes and mucous membranes may also contribute to overall exposure and may also invalidate the exposure standard. Aromatic hydrocarbons may produce sensitivity and redness of the skin. They are not likely to be absorbed into the body through the skin but branched species are more likely to.
Inhalation of aerosols (mists, fumes), generated by the material during the course of normal handling, may be harmful. There is some evidence to suggest that the material can cause respiratory irritation in some persons. The body's response to such irritation can cause further lung damage. Hydrogen sulfide poisoning can cause increased secretion of saliva, nausea, vomiting, diarrhea, giddiness, headache, vertigo, memory loss, palpitations, heartbeat irregularities, weakness, muscle cramps, confusion, sudden collapse, unconsciousness and death due to paralysis of breathing (at levels above 300 parts per million). The "rotten egg" odor is not a good indicator of exposure since odor fatigue occurs and odor is lost at over 200 ppm. The gas can enter the body through a punctured ear drum and even wearing some respiratory protection. Immediate supportive care is essential. Ensure medical help is addressed as part of the site emergency plan and that employees who may be accidentally exposed are made aware of the existence of such a plan. Inhalation of vapours may cause drowsiness and dizziness. This may be accompanied by narcosis, reduced alertness, loss of reflexes, lack of coordination and vertigo. Acute exposure to bitumen/ asphalt vapours may cause coughing, chest tightness, headache, muscle weakness, dizziness, tiredness, poor coordination, and even nausea and vomiting. Workers exposed to hot blown bitumens show bronchitis, rhinitis, oropharyngitis and laryngitis; symptoms include cough, phlegm, burning of the throat and chest, hoarseness, headache and nasal discharge. Guinea pigs, rabbits and mice exposed to blown bitumen fumes, aerosols and smoke, developed patchy regions of emphysema, bronchiolar dilation, pneumonitis, and severe localised bronchitis. Mice, exposed to aerosols of petroleum bitumens and smoke from heated petroleum bitumens, showed congestion, acute bronchitis, pneumonitis, bronchial dilation, abscess formation, epithelial atrophy, and necrosis. In health studies in the workplace, environmental measurement showed concentrations of asphalt, ranging from "non-detectable", where there was good mechanical ventilation, to 40 mg/m3, where there was very poor natural draft. Breathing zone samples, collected during drum-filling operations, ranged from 1.0 (upwind) to 5 mg/m3 (downwind) as means of 4-hour exposures. In the opinion of industrial hygienists conducting these studies, work conditions were satisfactory where asphalt fumes were kept below 10 mg/m3. Nerve damage can be caused by some non-ring hydrocarbons. Symptoms are temporary, and include weakness, tremors, increased saliva, some convulsions, excessive tears with discoloration and inco-ordination lasting up to 24 hours. If exposure to highly concentrated solvent atmosphere is prolonged this may lead to narcosis, unconsciousness, even coma and possible death.
Long term low level exposure to hydrogen sulfide may produce headache, fatigue, dizziness, irritability and loss of sexual desire. These symptoms may also result when exposed to hydrogen sulfide at high concentration for a short period of time. Limited evidence suggests that repeated or long-term occupational exposure may produce cumulative health effects involving organs or biochemical systems. There has been concern that this material can cause cancer or mutations, but there is not enough data to make an assessment. There is limited evidence that, skin contact with this product is more likely to cause a sensitization reaction in some persons compared to the general population. There is some evidence that human exposure to the material may result in developmental toxicity. This evidence is based on animal studies where effects have been observed in the absence of marked maternal toxicity, or at around the same dose levels as other toxic effects but which are not secondary non-specific consequences of the other toxic effects. Exposure to the material for prolonged periods may cause physical defects in the developing embryo (teratogenesis). Constant or exposure over long periods to mixed hydrocarbons may produce stupor with dizziness, weakness and visual disturbance, weight loss and anemia, and reduced liver and kidney function. Skin exposure may result in drying and cracking and redness of the skin. Chronic exposure to lighter hydrocarbons can cause nerve damage, peripheral neuropathy, bone marrow dysfunction and psychiatric disorders as well as damage the liver and kidneys. Oil may contact the skin or be inhaled. Extended exposure can lead to eczema, inflammation of hair follicles, pigmentation of the face and warts on the soles of the feet. Exposure to oil mists can cause asthma, pneumonia and scarring of the lungs. Oils have been linked to cancer of the skin and scrotum. Compounds that are less viscous and with smaller molecular weights are more dangerous. There may be liver damage and the lymph nodes may be affected; heart inflammation can also occur at high doses.