Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAME

WATTYL GLOSS STAIN AND VARNISH AEROSOL

NFPA

PRODUCT USE

Used according to manufacturer' s directions. Application is by spray atomization from a
hand held aerosol pack.

Section 2 - HAZARDS IDENTIFICATION

CANADIAN WHMIS SYMBOLS

EMERGENCY OVERVIEW

RISK

Irritating to eyes and respiratory system.
Extremely flammable.
Repeated exposure may cause skin dryness and cracking.
Vapors may cause dizziness or suffocation.
Risk of explosion if heated under confinement.

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED

  Not normally a hazard due to physical form of product.  Considered an unlikely route of entry in commercial/industrial environments.  Accidental ingestion of the material may be damaging to the health of the individual.  Considered an unlikely route of entry in commercial/industrial environments. The liquid may produce gastrointestinal discomfort and may be harmful if swallowed. Ingestion may result in nausea, pain and vomiting. Vomit entering the lungs by aspiration may cause potentially lethal chemical pneumonitis.  Ingestion of petroleum hydrocarbons can irritate the pharynx, esophagus, stomach and small intestine, and cause swellings and ulcers of the mucous. Symptoms include a burning mouth and throat; larger amounts can cause nausea and vomiting, narcosis, weakness, dizziness, slow and shallow breathing, abdominal swelling, unconsciousness and convulsions. Damage to the heart muscle can produce heart beat irregularities, ventricular fibrillation (fatal) and ECG changes. The central nervous system can be depressed. Light species can cause a sharp tingling of the tongue and cause loss of sensation there. Aspiration can cause cough, gagging, pneumonia with swelling and bleeding.  Central nervous system (CNS) depression may include general discomfort, symptoms of giddiness, headache, dizziness, nausea, anaesthetic effects, slowed reaction time, slurred speech and may progress to unconsciousness. Serious poisonings may result in respiratory depression and may be fatal.  

EYE

  This material can cause eye irritation and damage in some persons.  The material may produce severe irritation to the eye causing pronounced inflammation. Repeated or prolonged exposure to irritants may produce conjunctivitis.  

SKIN

  Skin contact with the material may damage the health of the individual; systemic effects may result following absorption.  Spray mist may produce discomfort.  Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine the skin prior to the use of the material and ensure that any external damage is suitably protected.  The material may cause skin irritation after prolonged or repeated exposure and may produce on contact skin redness, swelling, the production of vesicles, scaling and thickening of the skin.  

INHALED

  Inhalation may produce health damage*.  Inhalation of aerosols (mists, fumes), generated by the material during the course of normal handling, may be damaging to the health of the individual.  The material can cause respiratory irritation in some persons. The body's response to such irritation can cause further lung damage.  Material is highly volatile and may quickly form a concentrated atmosphere in confined or unventilated areas. Vapor is heavier than air and may displace and replace air in breathing zone, acting as a simple asphyxiant. This may happen with little warning of overexposure.  WARNING: Intentional misuse by concentrating/inhaling contents may be lethal.  Inhalation of vapours may cause drowsiness and dizziness. This may be accompanied by narcosis, reduced alertness, loss of reflexes, lack of coordination and vertigo.  Ketone vapors irritate the nose, throat and mucous membrane. High concentrations depress the central nervous system, causing headache, vertigo, poor concentration, sleep and failure of the heart and breathing. Some ketones can cause multiple nerve disorders, inducing "pins and needles" and weakness in the limbs.  Systemic effects of acetone inhalation exposure include central nervous system depression,  light-headedness, incoherent speech, ataxia, stupor, hypotension, tachycardia, metabolic acidosis, hyperglycaemia and ketosis. Rarely, convulsions and tubular necrosis may be evident. Other symptoms of exposure may include restlessness, headache, vomiting, low blood-pressure and rapid and irregular pulse, eye and throat irritation, weakness of the legs and dizziness. Inhalation of high concentrations may produce dryness of the mouth and throat, nausea, uncoordinated movement, loss of coordinated speech, drowsiness and, in severe cases, coma. Inhalation of acetone vapours over long periods causes irritation of the respiratory tract, coughing and headache. Rats exposed to 52200 ppm vapour for 1 hour showed clear signs of narcosis; fatalities occurred at 126600 ppm.  If exposure to highly concentrated solvent atmosphere is prolonged this may lead to narcosis, unconsciousness, even coma and possible death.  

CHRONIC HEALTH EFFECTS

  Principal route of occupational exposure to the gas is by inhalation.  Limited evidence suggests that repeated or long-term occupational exposure may produce cumulative health effects involving organs or biochemical systems.  Asthma-like symptoms may continue for months or even years after exposure to the material ceases. This may be due to a non-allergenic condition known as reactive airways dysfunction syndrome (RADS) which can occur following exposure to high levels of highly irritating compound. Key criteria for the diagnosis of RADS include the absence of preceding respiratory disease, in a non-atopic individual, with abrupt onset of persistent asthma-like symptoms within minutes to hours of a documented exposure to the irritant. A reversible airflow pattern, on spirometry, with the presence of moderate to severe bronchial hyperreactivity on methacholine challenge testing and the lack of minimal lymphocytic inflammation, without eosinophilia, have also been included in the criteria for diagnosis of RADS. RADS (or asthma) following an irritating inhalation is an infrequent disorder with rates related to the concentration of and duration of exposure to the irritating substance. Industrial bronchitis, on the other hand, is a disorder that occurs as result of exposure due to high concentrations of irritating substance (often particulate in nature) and is completely reversible after exposure ceases. The disorder is characterised by dyspnea, cough and mucus production.  There has been concern that this material can cause cancer or mutations, but there is not enough data to make an assessment.  There is some evidence that human exposure to the material may result in developmental toxicity. This evidence is based on animal studies where effects have been observed in the absence of marked maternal toxicity, or at around the same dose levels as other toxic effects but which are not secondary non-specific consequences of the other toxic effects.  Workers exposed to 700 ppm acetone for 3 hours/day for 7-15 years showed inflammation of the respiratory tract, stomach and duodenum, attacks of giddiness and loss of strength. Exposure to acetone may enhance liver toxicity of chlorinated solvents.  Repeated application of mildly hydrotreated oils (principally paraffinic), to mouse skin, induced skin tumors; no tumors were induced with severely hydrotreated oils.  Chronic solvent inhalation exposures may result in nervous system impairment and liver and blood changes. [PATTYS].