KOSLOW SOLN #11
Flammability | 0 | |
Toxicity | 2 | |
Body Contact | 4 | |
Reactivity | 0 | |
Chronic | 2 | |
SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4 |
Used as a reducing agent particularly in the manufacture of dyes, in tinning by galvanic
methods, in liquor finishing of wire, in sensitization of glass and plastics before
metallizing, as a soldering flux. Used as a mordant in dyeing with cochineal, in the
manufacture of tin chemicals, colour pigment, pharmaceuticals and sensitized paper, as a
lubricating oil additive, as a tanning agent, in removing ink stains, in yeast revivers,
as a reagent in analytical chemistry and as a catalyst in organic reactions.
"Soln. #11", Soln.#11, "Soln Number 11", "stannous chloride", "tin protochloride",
"stannous chloride, dihydrate", "stannous chloride, hydrated", "tin (II) chloride,
dihydrate", stannochlor, "tin salt", SnCl2.2H2O, Sn-Cl2-H4-O2
Harmful if swallowed.
Causes severe burns.
Risk of serious damage to eyes.
Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of less than 150 gram may be fatal or may produce serious damage to the health of the individual. The material can produce severe chemical burns within the oral cavity and gastrointestinal tract following ingestion. Tin salts are not very toxic. However, at high concentration, nausea, vomiting and diarrhea can occur. At very high levels growth may be affected.
The material can produce severe chemical burns to the eye following direct contact. Vapors or mists may be extremely irritating. If applied to the eyes, this material causes severe eye damage.
The material can produce severe chemical burns following direct contactwith the skin. The material may accentuate any pre-existing skin condition.
If inhaled, this material can irritate the throat andlungs of some persons. Persons with impaired respiratory function, airway diseases and conditions such as emphysema or chronic bronchitis, may incur further disability if excessive concentrations of particulate are inhaled. Exposure to fumes of tin oxide produce a benign pneumoconiosis without fibrosis or evidence of silicosis. The fact that there appears to be little respiratory disability is unexpected in view of the gross radiological abnormalities. Deposited particles are nodular and appear to be extracellular. No necrosis, foreign body giant-cell reaction or collagen formation is evident.
Principal routes of exposure are by accidental skin and eye contact andinhalation of generated dusts. Chronic exposure to tin dusts and fume can result in substantial amounts being deposited in the lungs and result in reduced lung function and difficulty breathing. Chronic exposure may cause liver and kidney damage.